Revision 44 for 'Calcific tendinitis'

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Calcific tendinitis

Calcific tendinitis (or calcific tendonitis) is a self-limiting condition due to deposition of calcium hydroxyapatite within tendons, usually of the rotator cuff. It is a common presentation of the hydroxyapatite crystal deposition disease (HADD)


Typically this condition affects middle-aged patients between the ages of 30 and 60, with a slight predilection for women 2.

Clinical presentation

The condition passes through four stages 2:

  1. pre-calcific
    • asymptomatic
    • fibrocartilaginous metaplasia (see below)
  2. calcific or formative
    • symptoms are variable from none to pain on movement
  3. resorptive
    • most symptomatic
    • pain due to extravasation of calcium hydroxyapatite into adjacent tissues, especially subacromial bursa, causing calcific bursitis
    • pain typically lasts 2 weeks
  4. post-calcific
    • variable symptomatology
    • some restriction of movement common
    • may last months


Calcific tendinitis results from the deposition of calcium hydroxyapatite within the substance of a tendon, and is thought to be due to decreased oxygen tension, leading to fibrocartilaginous metaplasia and secondary mineralization 1.


This condition most frequently affects the rotator cuff of the shoulder 1.

However, the condition may occur anywhere in the body with the hip and knee being the other most common locations 10.

Radiographic features

Plain radiograph

Calcific deposits are usually visualized as homogeneous hyperdensity with variable morphology, but typically globular/amorphous with poor margins.


Features of calcific tendinitis on ultrasound may include 7:

  • a curvilinear/ovoid calcification with acoustic shadowing
  • capsular soft tissue swelling
  • T1
    • hypointense homogeneous signal
    • adjacent tendon may be thickened
    • some enhancement surrounding deposit may be seen
  • T2
    • hypointense calcium deposits
    • hyperintense signal may be present peripherally due to edema
    • hyperintense subacromial-subdeltoid bursal fluid
  • T2*: calcifications may bloom

Treatment and prognosis

Controversial and difficult to measure due to the inherent variability of the symptoms and the self-limiting nature of the disease. Potential treatments include 2:

  • oral analgesic/anti-inflammatory medication
  • subacromial local anesthetic/steroid injection
  • aspiration of mineralized material
  • ultrasound therapy

Differential diagnosis

In the shoulder consider:

  • incidental calcification: seen in 2.5-20% of 'normal' healthy shoulders 1,2
  • degenerative calcification
    • seen in previously torn tendons
    • generally smaller
    • slightly older individuals
  • loose bodies
    • associated chondral defect
    • associated secondary osteoarthritis

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