Close
What is clinical triad of fat embolism syndrome?
Cerebral dysfunction, respiratory distress and skin petechiae.
The imaging findings are characteristic of those secondary to fat embolism.
-
extensive bilateral punctate foci of diffusion restriction, seen in both cerebral hemispheres. The most numerous foci are in the deep and periventricular white matter however further foci are seen involving the cortex, grey matter nuclei as well as the brainstem and cerebellar peduncles with only minimal involvement of the cerebellar hemispheres. Faint FLAIR and T2 signal is seen in these regions. No evidence of susceptibility artefact of T1 hyperintensity.
Diffuse axonal injury and ischaemia secondary to a hypotensive episode are considered much less likely.