Circumferential thickening of left ventricular walls. Focal increased trabeculations about the lateral apical wall, which does not meet the criteria for a noncompaction. Mild concentric thickening of right ventricular walls. Left atrial enlargement.
There is widespread delayed enhancement involving the myocardium and the subendocardium compatible with cardiac amyloidosis. The subendocardial enhancement involves both atria and ventricles.