Although there seems to be concentric left ventricular hypertrophy, this would be a misnomer as there is merely thickening of the LV wall due to amyloid deposition in the extracellular space. The myocardium itself is not hypertrophied. The amyloid results in a reduced capability of the heart to contract (restrictive pattern). 

Also note that there is reduced signal intensity of the blood pool due to pooling of contrast in the enlarged extracellular space of the ventricular walls.

Cardiac amyloidosis carries a very poor prognosis.