An ill-defined, T1 low and T2 high signal intensity lesion which is mildly suppressed on FLAIR is noted in the supra-sellar region extending into the intra-sellar area. There are a few, small, intra-lesional low signal intensities noted on FLAIR, suggestive of cystic spaces. DWI shows no diffusion restriction. On SWI, a few peripheral linear blooming artifacts could be appreciated which correspond to dark areas on SWI-Phase images, suggestive of calcifications. T1 fat sat postcontrast study shows avid enhancement of the solid components of the lesion with few, small, non-enhancing areas within the lesion denoting small cysts.
Anteriorly, it has involved the optic chiasm. Inferiorly, it appears to compress the pituitary gland. It is encasing the surrounding vessels including M1 segments of bilateral MCAs and A1 segments of bilateral ACAs. Postero-superiorly, it has involved the third ventricle leading to dilatation of bilateral lateral ventricles causing obstructive hydrocephalus.