Lateral pontine syndrome
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Lateral pontine syndrome, also known as Marie-Foix syndrome or Marie-Foix-Alajouanine syndrome, refers to one of the brainstem stroke syndromes which occurs due to occlusion of perforating branches of the basilar and anterior inferior cerebellar (AICA) arteries1,2. This results in infarction of the lateral aspect of the pons which produces a characteristic clinical picture from the involvement of the following pontine structures1,2:
- corticospinal tract: contralateral hemiplegia/hemiparesis.
- spinothalamic tracts: contralateral loss of pain and temperature sensation.
- cerebellar tracts: ipsilateral limb ataxia.
- facial nerve (CN VII) nucleus: ipsilateral facial paralysis.
- vestibulocochlear nerve (CN VIII) nuclei: ipsilateral hearing loss, vertigo and nystagmus.
History and etymology
The syndrome was first described by Pierre Marie (1853-1940), Charles Foix (1882-1927), and Théophile Alajouanine (1890-1980), French neurologists, in 1922 3.
-<p><strong>Lateral pontine syndrome</strong>, also known as <strong>Marie-Foix syndrome</strong>, refers to one of the <a href="/articles/brainstem-stroke-syndromes">brainstem stroke syndromes</a> which occurs due to occlusion of perforating branches of the <a href="/articles/basilar-artery">basilar</a> and <a href="/articles/anterior-inferior-cerebellar-artery">anterior inferior cerebellar (AICA) arteries</a>. This results in infarction of the lateral aspect of the pons which produces a characteristic clinical picture from the involvement of the following pontine structures:</p><ul>- +<p><strong>Lateral pontine syndrome</strong>, also known as <strong>Marie-Foix syndrome</strong> or <strong>Marie-Foix-Alajouanine syndrome</strong>, refers to one of the <a href="/articles/brainstem-stroke-syndromes">brainstem stroke syndromes</a> which occurs due to occlusion of perforating branches of the <a href="/articles/basilar-artery">basilar</a> and <a href="/articles/anterior-inferior-cerebellar-artery">anterior inferior cerebellar (AICA) arteries</a> <sup>1,2</sup>. This results in infarction of the lateral aspect of the pons which produces a characteristic clinical picture from the involvement of the following pontine structures <sup>1,2</sup>:</p><ul>
-<a title="Facial nerve" href="/articles/facial-nerve">facial nerve (CN VII)</a> nucleus: ipsilateral <a href="/articles/facial-palsy">facial paralysis</a>.</li>- +<a href="/articles/facial-nerve">facial nerve (CN VII)</a> nucleus: ipsilateral <a href="/articles/facial-palsy">facial paralysis</a>.</li>
-<a title="Vestibulocochlear nerve (CN VIII)" href="/articles/vestibulocochlear-nerve">vestibulocochlear nerve (CN VIII)</a> nuclei: ipsilateral hearing loss, vertigo and nystagmus.</li>-</ul>- +<a href="/articles/vestibulocochlear-nerve">vestibulocochlear nerve (CN VIII)</a> nuclei: ipsilateral hearing loss, vertigo and nystagmus.</li>
- +</ul><h4>History and etymology</h4><p>The syndrome was first described by <strong>Pierre Marie</strong> (1853-1940), <strong>Charles Foix</strong> (1882-1927), and <strong>Théophile Alajouanine</strong> (1890-1980), French neurologists, in 1922 <sup>3</sup>.</p>
References changed:
- 3. Marie P, Foix C, Alajouanine T. De I'atrophie cerebelleuse tardive a predominance corticale. Rev Neurol (Paris). 1922;29:1082-111.
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- cases
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