Transient global amnesia
Updates to Article Attributes
Transient global amnesia (TGA) is an anterograde and partial retrograde amnesia, usually of elderly people (>60 yearsolder age (50-70 year old), lasting lasting less than 24 hours without any other neurological or congestive symptoms. Most cases show complete resolution of symptoms within a few hours from onset of symptoms. Several hypothesis such as epileptic phenomena, stroke, or focal ischemia, etc, have been proposed with no consensus on the exact mechanism.1.
Radiographic assessment
MRI
ProspectiveBrain CT scan and conventional sequences of MRI brain show no abnormalities. Prospective and retrospective studies based on small number of patients show diffusion weighted imaging (DWI) can detect small punctate hyperintense lesion(s) in the CA1 area of the hippocampus in patients with TGA.2-4.Typical appearance is small punctate hyperintense focus on the lateral edge of the hippocampus gyrus abutting the temporal horn and can be bilateral and even multifocal.2-4
-<p><strong>Transient global amnesia (TGA) </strong>is an anterograde and partial retrograde amnesia, usually of elderly people (>60 years), lasting less than 24 hours. Most cases show complete resolution of symptoms within a few hours from onset of symptoms. Several hypothesis have been proposed with no consensus on the exact mechanism.<sup>1</sup></p><h4><strong>Radiographic assessment</strong></h4><h5><strong>MRI</strong></h5><p>Prospective and retrospective studies based on small number of patients show diffusion weighted imaging (DWI) can detect small punctate hyperintense lesion(s) in the hippocampus in patients with TGA.<sup>2-4</sup> <br><br>Typical appearance is small punctate hyperintense focus on the lateral edge of the hippocampus gyrus abutting the temporal horn and can be bilateral and even multifocal.<sup>2-4</sup> </p>- +<p><strong>Transient global amnesia (TGA) </strong>is an anterograde and partial retrograde amnesia, usually of older age (50-70 year old) lasting less than 24 hours without any other neurological or congestive symptoms. Most cases show complete resolution of symptoms within a few hours from onset of symptoms. Several hypothesis such as epileptic phenomena, stroke, or focal ischemia, etc, have been proposed with no consensus on the exact mechanism <sup>1</sup>.</p><h4><strong>Radiographic assessment</strong></h4><h5><strong>MRI</strong></h5><p>Brain CT scan and conventional sequences of MRI brain show no abnormalities. Prospective and retrospective studies based on small number of patients show diffusion weighted imaging (DWI) can detect small punctate hyperintense lesion(s) in the CA1 area of the hippocampus in patients with TGA <sup>2-4</sup>.<br><br>Typical appearance is small punctate hyperintense focus on the lateral edge of the hippocampus gyrus abutting the temporal horn and can be bilateral and even multifocal.<sup>2-4</sup> </p>
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