Hypoglycemic encephalopathy
Updates to Case Attributes
The cause was suicide attempt by overdosage of insulin in a patient who has psychiatric disorders and is unknown to be diabetic.
HypoglycemicHypoglycaemic brain injury is typically diagnosed in comatose adult diabetic men on insulin with brain insult due to insulin overdosage. The lesion usually involves the frontal, parietal, temporal and occipital cortex and subcortex with relative sparing of the deep white matter. The basal ganglia are involved in severe cases. The thalami are always spared as well as the cerebellum and the brain stem.
The lack of deep white matter injury, haemorrhagic reperfusion as well as the lack of the thalamic affection are the main differentiating points between hypoglycemichypoglycaemic and hypoxic-ischemic-ischaemic brain insult.
-<p>The cause was suicide attempt by overdosage of insulin in a patient who has psychiatric disorders and is unknown to be diabetic.</p><p><a href="/articles/hypoglycaemic-encephalopathy">Hypoglycemic brain injury</a> is typically diagnosed in comatose adult diabetic men on insulin with brain insult due to insulin overdosage. The lesion usually involves the frontal, parietal, temporal and occipital cortex and subcortex with relative sparing of the deep white matter. The basal ganglia are involved in severe cases. The thalami are always spared as well as the cerebellum and the brain stem. </p><p>The lack of deep white matter injury, haemorrhagic reperfusion as well as the lack of the thalamic affection are the main differentiating points between hypoglycemic and <a href="/articles/hypoxic-ischaemic-brain-injury-1">hypoxic-ischemic</a> brain insult. </p>- +<p>The cause was suicide attempt by overdosage of insulin in a patient who has psychiatric disorders and is unknown to be diabetic.</p><p><a href="/articles/hypoglycaemic-encephalopathy">Hypoglycaemic brain injury</a> is typically diagnosed in comatose adult diabetic men on insulin with brain insult due to insulin overdosage. The lesion usually involves the frontal, parietal, temporal and occipital cortex and subcortex with relative sparing of the deep white matter. The basal ganglia are involved in severe cases. The thalami are always spared as well as the cerebellum and the brain stem. </p><p>The lack of deep white matter injury, haemorrhagic reperfusion as well as the lack of the thalamic affection are the main differentiating points between hypoglycaemic and <a href="/articles/hypoxic-ischaemic-brain-injury-1">hypoxic-ischaemic</a> brain insult. </p>
Updates to Study Attributes
Image MRI (T2) ( update )
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