Pericardial agenesis
Updates to Study Attributes
Heart in levoposition with the apex positioned posteriorly. Contrast can be see entering the left atrium from the pulmonary veins, passing into the left ventricle, and leaving through the ascending aortic graft (sternotomy wires also present). Thrombus can be see in the ascending aorta.
Updates to Study Attributes
Heart in levoposition with the apex positioned posteriorly. Contrast can be see entering the left atrium from the pulmonary veins, passing into the left ventricle, and leaving through the ascending aortic graft (sternotomy wires also present). Thrombus can be see in the ascending aorta.
Updates to Study Attributes
Heart in levoposition with the apex positioned posteriorly. Contrast can be see entering the left atrium from the pulmonary veins, passing into the left ventricle, and leaving through the ascending aortic graft (sternotomy wires also present). Thrombus can be see in the ascending aorta. Hypoplastic left lower lobe.
Image CT (C+ arterial phase) ( update )
Image CT (C+ arterial phase) ( update )
Image 2 CT (C+ arterial phase) ( create )
Image 3 CT (C+ arterial phase) ( create )
Updates to Case Attributes
The patient was later placed on anticoagulation for treatment of the thrombus with follow-up scheduled in a month's3 months time to assess for resolution.
Pericardial agenesis is caused by defective development of the pleuropericardial membranes. Congenital pericardial agenesis is usually asymptomatic and discovered incidentally. Patients may present with a right bundle branch block on EKG, poor R wave progression, and prominent P waves. Complete pericardial agenesis has the best prognosis whereas partial agenesis may leave opportunity for herniation of tissues. Intervention should only be considered if complications of the defect are present.
This case was submitted with supervision and input from:
M. Attrach, M.D.Cardiovascular RadiologistErasmus Medical CenterRotterdam, The Netherlands
-<p>The patient was later placed on anticoagulation for treatment of the thrombus with follow-up scheduled in a month's time to assess for resolution.</p><p>Pericardial agenesis is caused by defective development of the pleuropericardial membranes. Congenital pericardial agenesis is usually asymptomatic and discovered incidentally. Patients may present with a right bundle branch block on EKG, poor R wave progression, and prominent P waves. Complete pericardial agenesis has the best prognosis whereas partial agenesis may leave opportunity for herniation of tissues. Intervention should only be considered if complications of the defect are present.</p><p>This case was submitted with supervision and input from:</p><p>M. Attrach, M.D.<br>Cardiovascular Radiologist<br>Erasmus Medical Center<br>Rotterdam, Netherlands</p>- +<p>The patient was later placed on anticoagulation for treatment of the thrombus with follow-up scheduled in 3 months time to assess for resolution.</p><p>Pericardial agenesis is caused by defective development of the pleuropericardial membranes. Congenital pericardial agenesis is usually asymptomatic and discovered incidentally. Patients may present with a right bundle branch block on EKG, poor R wave progression, and prominent P waves. Complete pericardial agenesis has the best prognosis whereas partial agenesis may leave opportunity for herniation of tissues. Intervention should only be considered if complications of the defect are present.</p><p>This case was submitted with supervision and input from:</p><p>M. Attrach, M.D.<br>Cardiovascular Radiologist<br>Erasmus Medical Center<br>Rotterdam, The Netherlands</p>
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